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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. vimax penis enlargement technique vimax homemade penis enlargement penile enlargement surgeries buy penis enlargment pills best penis elargement penis enlarement cream penis enlarement stretcher magnarx

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It might benefit all men to know and understand that erection weakness, as well as premature ejaculation are essentially coded messages from your body needing to be deciphered and understood. The statistics show, and is supported by my findings through contacting members of my penis enlargement exercise website, that most of those suffering from premature ejaculation are actually younger men, who have less control over their bodies and less experience than those of an older man. In fact, erectile dysfunction and premature ejaculation were the most frequently reported sexual concerns according to 9536 men who connected with a male health helpline. While the diagnosis and treatment of the sometimes deep-rooted psychological factors manifesting themselves in the form of premature ejaculation are beyond the scope of this article, what is the scope of this article is the physical treatment of the condition. Erectile dysfunction and premature ejaculation are the most common male sexual disorders and each may have various degrees of severity. Some cases of premature ejaculation are so severe, the man will reach orgasm as soon as his penis makes vaginal contact. This is because two major causes of premature ejaculation are over-eagerness and anxiety. It seems clear that a lot of men who suffer from premature ejaculation are embarrassed to talk about it, even with their doctors. Some of the physical causes of premature ejaculation are diabetes, a tight foreskin, and disorders of the nervous system. Depression may also lead to erectile dysfunction by creating a loss of sex drive, or by the medications used for treatment of the condition. As with other types of sexual dysfunction, there has never been consensus about what exactly constitutes rapid or premature ejaculation. A Possible Treatment Researchers at the University of Minnesota, have shown that the drug dapoxetine is a safe and effective drug treatment for premature ejaculation. In addition a University of Alberta researcher is very close to finding a possible additional promising treatment for premature ejaculation. Until Dapoxetine will be approved by FDA, men need to know there does exist an online program available for possible premature ejaculation treatment, and it's available at my website. If however, you are looking for brick-and-morter treatment options, those possibilities are: Therapy Counseling Medication Combination of these methods. Hypnosis has also proven very effective in the treatment of premature ejaculation in a very small number of men. Many common 'solutions' to prevent premature ejaculation are completely counterproductive. As sexual disorders like premature ejaculation are becoming common around the globe, the Universities will continue to churn out possible medications to treat it with. What they've overlooked, is that most men who suffer with premature ejaculation are actually just in need of a little reprogramming...and a penis exercise program. penis enhancement excercises penis enlargment before and after enhancement free penis pills sample penis enhancement system penis enlargment patch natural penis enlargment pills natural pnis enlargement magna rx pills penis enlargement secret

There are four kinds of parasites The intestines are sometimes infested by parasites, which trouble the patient. The most commonly noticed parasites in India are: threadworm, tapeworm, hookworm and the roundworm. They exist in the intestines because they find a congenial climate for their growth which again is the result of wrong type of feeding which most people indulges in. patients suffering from these worms generally acquire a voracious appetite, but in spite of massive quantities of food they take in, there is no gain the weight. Sometimes they are pale and sickly and, therefore, gloomy. Infants and young children are likely to be irritated all the time and are prone to throw tantrums at the slightest excuse. The most common and the least harmful parasite infesting the intestines is the threadworm, or enterobius vermicularis. When they are expelled from the anus, they tend to create uncontrollable itch and the patient is forced to scratch his anus. They are small, thread like creatures, which can be noticed in the stools of children. The hookworm and the tapeworm reassemble earthworms: they are, fortunately, less common in our country than threadworms. Causes It is the dirty habits, like eating food without washing your hands, putting the finger first in the nostril then in the mouth, eating contaminated food, scratching the anus and then eating food without thoroughly washing your hands, using underwear worn by a person suffering from threadworms, constipation, and mucus in the stools, which encourage the parasites to infest the intestines. Treatment Traditional medicine depends on drugs to kill the parasites; they may give temporary relief, but that is more likely to harm the digestive processes and create other complications. Naturopathy, on the other hand, aims at strengthening the intestines so that they can purge themselves of the parasites. The stools of a child suspected to be suffering from threadworms could be examined visually: in some cases they would be found sticking to the anus of the child. Treatment of threadworm-infested patient must start with cleanliness. A child suffering from it should be administered a warm water enema to which juice of half a fresh lime has been added. It should be followed by administration of about 50 to 100 militres of coconut oil with the help of a syringe through the anus. The child should be asked to strain himself at the time of passing the stools. The best course would be to start the treatment with fasting: if the young patient or his mother cavils at it, he should be administered fruit juice or clear vegetables soup mixed with water. Lukewarm enema must be given during the treatment both in the morning and evening. Enema would help expel mucus and threadworms from the intestines. Bedclothes of the patient must be aired in the sun every day and he should stay in a well-ventilated room. If the child is walking he should be encouraged to fast for two days followed by a diet of fruits and boiled vegetables for five to six days. Raw vegetables like carrot, cucumber and onions can be given in addition to tomatoes. Mil and cereals should be totally avoided. Potatoes roasted over the coals and raisins soaked in water could also be given. After that the patient could be allowed porridge or gruel. When he has started taking cereals, milk should not be given to him not lentils. Cold compresses and mudpack applied from the navel to the pubes can also help in the initial stages of the treatment. Juice of garlic could also be taken recourse to since it kills the parasites without harming the patient. This course of treatment could help in dealing with disease like the enlargement of a spleen, hepatitis low fever, goiter, mumps, discharge of pus from the ear and swollen eyes in children. For more information regarding Home Remedies for Parasites, Herbal Remedy visit http://www.natural-homeremedies.com penis enlarement information vimax cheap penis enlargement vimax male virility sexual enhancement penis enlarement testimonials penis enlargement secret penis enlagement exercise male penis enhancement com enlargement penis penis pump penis enlargement secret

Sexual dysfunction, in one form or the other and in varying degrees, is common among both men and women. According to recent studies, a large percentage of all men and women encounter some sort of sexual dysfunction at some point in their lives. And as they grow older, such problems become increasingly common. In males, sexual dysfunction may be of different types like lack of desire, failure to obtain and/or maintain an erection, and other problems like premature ejaculation and ejaculatory impotence, or the inability to ejaculate in coitus. Erectile dysfunction, however, is certainly the cause for maximum concern. For the treatment of erectile dysfunction, three oral medications are available: sildenafil (Viagra), vardenafil (Levitra), and tadalafil (Cialis). They boost the levels of nitric oxide, thereby relaxing the blood vessels and smooth muscle in the penis. As a result, the flow of blood is increased, and erection is achieved and maintained. Whatever may be the cause of erectile dysfunction, sildenafil, vardenafil, and tadalafil have proved themselves extremely helpful. In Europe, another drug under the brand name of Uprima (apomorphine) has hit the market, although it still awaits the approval of the U.S. FDA. Instead of increasing blood flow in the penis, apomorphine acts on the brain to enhance erection. These drugs should not, however, be used by those who have had a heart problem during the past six months, or those with serious liver or kidney ailments, certain eye disorders, and extreme levels of blood pressure. In females, lack of libido, failure to become aroused, lack of orgasm or anorgasmy, and vaginismus are the common sexual dysfunctions. Although no medications have yet been approved specifically for the treatment of female sexual dysfunction, research is continuing on the subject, which includes looking into the possibility of the use of sildenafil in females. A pharmaceutical major is now about to get the go-ahead for a testosterone patch for the treatment of low libido in postmenopausal women. Falls in testosterone levels are believed to be responsible to a large extent for lack of libido in both men and women. The proposed transdermal testosterone patch, to be marketed under the name “Intrinsa,” is worn on the lower abdomen. Further research will determine who should or shouldn’t use the testosterone patch, and its possible side effects as well. free penis enlagement tip vimax penis pillss in uk cheap penis enlarement pills penis enargement product penis enlargement review truth about penis enlagement penis elargement pills product vimax penis enlargement video penis enlargement secret

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